Formula 9 Hardcore Dieting Pill 60ct by Dynakor

Formula 9 is a new breed Of Energy and Weight management pill! A newly discovered, natural compound has been found that offers substantial benefits for overweight consumers. This patented plant extract is found exclusively in Formula 9, along with a proprietary mixture of additional energy- and weight-loss promoting ingredients… which have been specifically formulated to deliver an entirely new breed of powerful and effective ergogenic diet aids. The Study: Patented Plant Extract Induces Weight Loss and Increases the Lean to Fat Mass Ratio in Humans Central to the effectiveness of Formula 9 is the presence of the new weight-loss agent. In a recent randomized, double-blind, placebo-controlled trial, 50 volunteers with a Body Mass Index (BMI) over 25 (moderately to severely obese) were randomized to two groups receiving either placebo capsules or a capsules containing 200 mg of the novel standardized extract (called CAE) found in Formula 9. Each volunteer took one capsule with each main meal, two a day, for 60 days. All subjects were instructed to follow a lowcalorie diet plan and not to change any of their physical activities. At the end of the 60- day study, the treatment group experienced a mean reduction in weight of about 11 pounds (5.7%) that was highly statistically different from the smaller weight reduction experienced by the placebo group (only 5.4 pounds (2.9%)(p<0.00001). Consequently, the BMI decreased significantly in the experimental CAE group compared to the placebo group. Moreover, the muscle mass/fat mass ratio increased significantly in the treatment group compared to the control group: 2.03 (4%) vs. 0.6 (1.5%) respectively (p<0.013). The sizeable decrease of body weight, BMI, and fat mass observed in the treatment group demonstrates that the Formula 9 CAE is able to significantly intensify weight loss in a group of volunteers ingesting a low-caloric diet. (The object of the lowcaloric diet was to lower the carbohydrate load, which would account for the modest weight loss seen in the placebo group.) The additional, substantial weight loss is postulated to be due to the ability of CAE to inhibit both glucose absorption and the activity of the enzyme glucose-6-phosphatase. The latter activity would limit the release of glucose into the general circulation and therefore limit insulinemia, which would in turn limit fat storage and new body fat formation and lead to utilization of the fat reserves for energy. How CAE Works Proper glucose metabolism is fundamental to effective weight management. Maintaining a normal blood glucose concentration is a finely balanced equilibrium between food intake, energy expenditure and the mechanisms by which glucose is stored or, is released into the blood. The human clinical study using the standardized, highly purified CAE targeted weight loss via an activity on postprandial blood glucose concentration (PBGC). PBGC is the fancy term referring to the amount of sugar remaining in your blood after eating a meal (the italicized terms are synonymous). This figure varies according to the composition of the meal but in every case is accompanied by an increase in the secretion of insulin and a decrease in the synthesis of glucagon. This hormonal activity results in the decrease of blood sugar by increasing its storage as glycogen in the liver or muscles (and in the form of fat (triglycerides) in fat cells, if glycogen storage areas fill up). This is called the 'absorption phase,' as the sugar is being absorbed from the blood. Over time (toward the end of the absorption phase or when fasting), the plasma glucose level decreases and reversal of the hormonal regulation occurs such that insulin levels drop and the secretion of glucagon increases. This activity results in the release of muscle glycogen for use as energy. High levels of glucagon will act to produce fatty acids from stored fat. The Role of Chlorogenic Acid in CAE Chlorogenic acids are a group of esters of varying composition. The most common of these esters is 5-caffeoylquinic acid. It is commonly called "chlorogenic acid" in research papers, but this term may also be used to refer to other esters in the mix found in CAE. Hence, for the sake of simplicity, the term chlorogenic acid (CA) will be used in this paper as a generic term for all members of the ester group. If the results of the study are indicative, the presence of CA in the CAE increased the effectiveness of reduced GI by a significant margin. This was not an unexpected outcome. Rather, it was a reasonable and predicted result, based on the observations of a number of scientific papers describing the action of CA on a number of related parameters. One of the most important of those actions is a postprandial reduction in the absorption of glucose from the intestines. Decreased glucose absorption means less excess glucose in the blood, an eventual decrease in insulin resistance, a propensity for greater fat burning activity in liver and muscle cells, reduced fat storage and loss of body weight. The specific mode of action is an inhibitory effect on the sodium ion electrochemical gradient which draws glucose into the cells (enterocytes) lining the gut. This appears to be a dose-dependant phenomenon (in rats the effect has been as high as 80%). In a human study it was found that CA significantly attenuated the postprandial release of GIP (glucose-dependent insulinotropic polypeptide). The GIP response is directly related to the amount of glucose absorbed at the intestinal barrier; hence, a decrease in GIP reflects a decrease in glucose absorption. Another manner in which CA decreases the blood glucose load is through the inhibition of an enzyme known as glucose-6-phosphatase (G6P). G6P plays an important role in the homeostatic control of blood sugar concentration. Present only in the liver, it is really an enzyme system and is responsible for the conversion of glucose-6-phosphate into glucose capable of passing into the general circulation. Inhibition of G6P causes a reduction in the liver's production of glucose and consequently decreases abnormally high levels of glucose in the blood. All sugars from the diet are transformed into G6P, which will be stored as glycogen because they cannot be broken down into glucose and released into the blood by G6P. Animal studies indicate that CA improves sensitivity to insulin by improving the distribution of minerals and by reducing peripheral insulin resistance. To summarize the body of work done on chlorogenic acid, the following sequence of events will occur following its regular consumption: A reduction in the intestinal absorption of glucose. Inhibition of glucose-6-phosphatase (G6P). Inhibition of the release of hepatic glucose originating from: the diet glycogenolyis (the creation of glucose from glycogen) gluconeogenesis (the formation of glucose, especially by the liver, from noncarbohydrate sources, such as amino acids and the glycerol portion of fats). Lowering of blood glucose concentration. A decrease in the secretion of insulin and an increase in the secretion of glucagon. Mobilization of fat reserves to supply energy required by muscles and other tissues. The burning of free fatty acids for the production of energy in cellular energy cycles. An Important Note About CAE Extraction In order to produce this extract with proven weight-reducing activity, the raw material must undergo considerable processing. The desired end result is a product standardized to specific levels of chlorogenic acids and 5-caffeoylquinic acid. However, proper, safe CAE extraction requires and additional processing step: the removal of potentially harmful diterpines. Until this crucial additional processing step was accomplished (and we know of only one manufacturing who takes this important last step), we had not considered CAE suitable for long-term human consumption. Now that a diterpene-free extract is availa

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